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Epigenetic regulation, such as DNA methylation and non-coding RNAs, has also been identified to modulate key proteins in both ferroptosis and cuproptosis, offering new perspectives for co-targeting (81, 82)

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Apart from the receptor-dependent hormone signaling, certain metabolites of estrogens act as direct carcinogens, such as the catechol 4-hydroxyestradiol and its derivative quinones [148]