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Recently, it was shown that recombinant human GSTs, in particular GSTP1, significantly contributed to the inactivation of AQ-derived QIs by catalyzing GSH-conjugation (Zhang et al., 2017a) ( Figure 1 )

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However, it has not been established whether the fructose-2,6-bisphosphatase TIGAR (Tp53-induced Glycolysis and Apoptosis Regulator), which promotes flux through the PPP and interacts with HKII under hypoxic conditions [77], promotes preferential PPP-flux in neurons and thereby cooperates with HKII to prevent cell death