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There is a hypothesis that a deficit of GABA, whose transmission occurs via GABA receptors, is the primary cause of cortical circuit dysfunction in schizophrenia, while cognitive deficits result from altered gamma oscillation circuitry [107]
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Levels of pro-inflammatory cytokines, such as interleukin 1 β (IL-1β), IL-2, and IL-8, and TNF-α in plasma are significantly increased in asymptomatic patients compared to symptomatic patients, which are positively related to plasma C26:0
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