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It drives neuronal mitochondrial biogenesis through CREB-PGC-1α signaling, a pathway that can bolster cellular resilience to stress

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This implies a potential negative feedback loop whereby “expression of MC3R by POMC neurons provides a potential circuit for amplification of AgRP-mediated signals, because AgRP-induced inhibition of POMC neurons via the MC3R would reinforce the postsynaptic effects of AGRP