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The supportive mechanistic data are largely rodent and cell-based, and the β3-adrenergic pathway that appears to contribute in mice is expressed and regulated differently in human adipose tissue

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The survival and activation/proliferation of OPCs, as well as and their enhanced and continuous differentiation into Enpp6 -positive newly-forming/formed OLs, despite the late onset (from 2-3 weeks and beyond) demyelination under the continuous pressure of CPZ, underpinned a strong potential for remyelination after CPZ withdrawal